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Cited 133 time in webofscience Cited 151 time in scopus
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dc.contributor.authorKong, YY-
dc.contributor.authorBoyle, WJ-
dc.contributor.authorPenninger, JM-
dc.date.accessioned2016-03-31T13:26:25Z-
dc.date.available2016-03-31T13:26:25Z-
dc.date.created2009-02-28-
dc.date.issued2000-10-
dc.identifier.issn0167-5699-
dc.identifier.other2000-OAK-0000001584-
dc.identifier.urihttps://oasis.postech.ac.kr/handle/2014.oak/19829-
dc.description.abstractThe TNF-family molecule osteoprotegerin ligand (OPGL, also known as TRANCE, RANKL and ODF) is a key regulator of bone remodeling and essential for the development and activation of osteoclasts. Intriguingly, OPGL also regulates T cell-dendritic cell communications, dendritic cell survival and lymph-node organogenesis. Here, Young-Yun Kong and colleagues discuss the role of OPGL, and its receptor RANK, in the immune system and in bone metabolism.-
dc.description.statementofresponsibilityX-
dc.languageEnglish-
dc.publisherELSEVIER SCI LTD-
dc.relation.isPartOfIMMUNOLOGY TODAY-
dc.subjectTUMOR-NECROSIS-FACTOR-
dc.subjectPERIPHERAL LYMPHOID ORGANS-
dc.subjectOSTEOCLASTOGENESIS-INHIBITORY FACTOR-
dc.subjectLYMPHOTOXIN BETA-RECEPTOR-
dc.subjectAPOPTOSIS-INDUCING LIGAND-
dc.subjectTNF FAMILY MEMBER-
dc.subjectT-CELLS-
dc.subjectRHEUMATOID-ARTHRITIS-
dc.subjectDENDRITIC CELLS-
dc.subjectDEFICIENT MICE-
dc.titleOsteoprotegerin ligand: a regulator of immune responses and bone physiology-
dc.typeArticle-
dc.contributor.college생명과학과-
dc.identifier.doi10.1016/S0167-5699(00)01718-7-
dc.author.googleKong, YY-
dc.author.googleBoyle, WJ-
dc.author.googlePenninger, JM-
dc.relation.volume21-
dc.relation.issue10-
dc.relation.startpage495-
dc.relation.lastpage502-
dc.relation.journalIMMUNOLOGY TODAY-
dc.relation.indexSCI급, SCOPUS 등재논문-
dc.relation.sciSCI-
dc.collections.nameJournal Papers-
dc.type.rimsART-
dc.identifier.bibliographicCitationIMMUNOLOGY TODAY, v.21, no.10, pp.495 - 502-
dc.identifier.wosid000089991200007-
dc.date.tcdate2019-01-01-
dc.citation.endPage502-
dc.citation.number10-
dc.citation.startPage495-
dc.citation.titleIMMUNOLOGY TODAY-
dc.citation.volume21-
dc.contributor.affiliatedAuthorKong, YY-
dc.identifier.scopusid2-s2.0-0033791573-
dc.description.journalClass1-
dc.description.journalClass1-
dc.description.wostc112-
dc.type.docTypeArticle-
dc.subject.keywordPlusTUMOR-NECROSIS-FACTOR-
dc.subject.keywordPlusPERIPHERAL LYMPHOID ORGANS-
dc.subject.keywordPlusOSTEOCLASTOGENESIS-INHIBITORY FACTOR-
dc.subject.keywordPlusLYMPHOTOXIN BETA-RECEPTOR-
dc.subject.keywordPlusAPOPTOSIS-INDUCING LIGAND-
dc.subject.keywordPlusTNF FAMILY MEMBER-
dc.subject.keywordPlusT-CELLS-
dc.subject.keywordPlusRHEUMATOID-ARTHRITIS-
dc.subject.keywordPlusDENDRITIC CELLS-
dc.subject.keywordPlusDEFICIENT MICE-
dc.relation.journalWebOfScienceCategoryImmunology-
dc.description.journalRegisteredClassscie-
dc.description.journalRegisteredClassscopus-
dc.relation.journalResearchAreaImmunology-

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공영윤KONG, YOUNG YUN
Dept of Life Sciences
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